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Cleveland Clinic Journal of Medicine Jun 2022
Topics: Humans; Pulmonary Edema
PubMed: 35649558
DOI: 10.3949/ccjm.89c.06002 -
American Journal of Therapeutics 2018Acute carbon monoxide (CO) poisoning is the most common cause of poisoning and poisoning-related death in the United States. It manifests as broad spectrum of symptoms... (Review)
Review
Acute carbon monoxide (CO) poisoning is the most common cause of poisoning and poisoning-related death in the United States. It manifests as broad spectrum of symptoms ranging from mild headache, nausea, and fatigue to dizziness, syncope, coma, seizures resulting in cardiovascular collapse, respiratory failure, and death. Cardiovascular complications of CO poisoning has been well reported and include myocardial stunning, left ventricular dysfunction, pulmonary edema, and arrhythmias. Acute myocardial ischemia has also been reported from increased thrombogenicity due to CO poisoning. Myocardial toxicity from CO exposure is associated with increased short-term and long-term mortality. Carboxyhemoglobin (COHb) levels do not correlate well with the clinical severity of CO poisoning. Supplemental oxygen remains the cornerstone of therapy for CO poisoning. Hyperbaric oxygen therapy increases CO elimination and has been used with wide variability in patients with evidence of neurological and myocardial injury from CO poisoning, but its benefit in limiting or reversing cardiac injury is unknown. We present a comprehensive review of literature on cardiovascular manifestations of CO poisoning and propose a diagnostic algorithm for managing patients with CO poisoning.
Topics: Algorithms; Biomarkers; Carbon Monoxide Poisoning; Carboxyhemoglobin; Heart Diseases; Humans; Hyperbaric Oxygenation; Myocardial Stunning; Practice Guidelines as Topic; Pulmonary Edema; Severity of Illness Index; United States
PubMed: 24518173
DOI: 10.1097/MJT.0000000000000016 -
Pediatric Emergency Care Mar 2018Reexpansion pulmonary edema is a rare complication that may occur after drainage of pneumothorax or pleural effusion. A number of factors have been identified that... (Review)
Review
Reexpansion pulmonary edema is a rare complication that may occur after drainage of pneumothorax or pleural effusion. A number of factors have been identified that increase the risk of developing reexpansion pulmonary edema, and pathophysiologic mechanisms have been postulated. Patients may present with radiographic findings alone or may have signs or symptoms that prompt evaluation and diagnosis. Clinical presentations range from mild cough to respiratory failure and hemodynamic compromise. Treatment strategies are supportive, and should be tailored to match the severity of the condition.
Topics: Child; Drainage; Female; Humans; Lung; Pediatrics; Pleural Effusion; Pneumothorax; Pulmonary Atelectasis; Pulmonary Edema; Risk Factors
PubMed: 29494460
DOI: 10.1097/PEC.0000000000001435 -
Current Opinion in Critical Care Aug 2014Early acute respiratory distress syndrome (ARDS) is characterized by protein-rich inflammatory lung edema often associated with a hydrostatic component. Mechanical... (Review)
Review
PURPOSE OF REVIEW
Early acute respiratory distress syndrome (ARDS) is characterized by protein-rich inflammatory lung edema often associated with a hydrostatic component. Mechanical ventilation with positive intrathoracic pressure further induces salt and water retention, while impairing the pathways designed for edema clearance. In this framework, we will review the recent findings on fluid strategy and edema clearance in ARDS.
RECENT FINDINGS
Consistently, conservative strategies lead to better oxygenation and reduce the length of mechanical ventilation. A possible drawback associated with conservative strategy is the impaired cognitive function. Echography may be used for safer use of furosemide or hemofiltration therapy during edema clearance. Albumin and furosemide techniques may accelerate edema clearance, particularly when pulmonary capillary permeability is restored. Beta-2 agonist therapy does not accelerate edema clearance and is potentially dangerous.
SUMMARY
Lung edema is likely the single pathogenic factor more relevant for ARDS severity and outcome. Fluid overload must be avoided. Several monitoring techniques are available to reach this target. No specific studies are available to recommend a given fluid composition in ARDS. In our opinion, the general recommendations for fluid composition suggested for severe sepsis and septic shock should be applied to ARDS that may be considered an organ-confined sepsis.
Topics: Fluid Therapy; Humans; Pulmonary Edema; Respiratory Distress Syndrome
PubMed: 24979554
DOI: 10.1097/MCC.0000000000000105 -
The American Journal of Medicine Mar 2022
Topics: Humans; Pulmonary Edema
PubMed: 34793749
DOI: 10.1016/j.amjmed.2021.10.022 -
Nursing Standard (Royal College of... Jun 2016This informative CPD article on acute pulmonary oedema has improved my knowledge of the condition.
This informative CPD article on acute pulmonary oedema has improved my knowledge of the condition.
Topics: Dyspnea; Humans; Nursing Assessment; Pulmonary Edema; United Kingdom
PubMed: 27275916
DOI: 10.7748/ns.30.40.61.s46 -
AACN Advanced Critical Care 2018Pulmonary edema is an acute pregnancy complication that, if uncorrected, can result in increased maternal and fetal morbidity and mortality. Although pulmonary edema is... (Review)
Review
Pulmonary edema is an acute pregnancy complication that, if uncorrected, can result in increased maternal and fetal morbidity and mortality. Although pulmonary edema is relatively rare in the general obstetrics population, pregnant patients are at increased risk for pulmonary edema because of the physiologic changes of pregnancy. The risk may be exacerbated by certain pregnancy-related diseases, such as preeclampsia. Prompt identification and appropriate clinical management of pulmonary complications is critical to prevent adverse outcomes in pregnant patients. This article reviews the collaborative treatment of pulmonary edema in pregnant women with complex critical illnesses.
Topics: Critical Care Nursing; Female; Humans; Obstetric Nursing; Practice Guidelines as Topic; Pregnancy; Pregnancy Complications; Pulmonary Edema
PubMed: 30185499
DOI: 10.4037/aacnacc2018958 -
Journal of Forensic and Legal Medicine Feb 2019Strongyloidiasis is an infectious disease affecting approximately 30-100 million people globally. The main human pathogen is Strongyloides stercoralis which may cause a... (Review)
Review
Strongyloidiasis is an infectious disease affecting approximately 30-100 million people globally. The main human pathogen is Strongyloides stercoralis which may cause a brief period of acute symptoms and signs after the initial infection, and then lapse into a chronic asymptomatic carrier state for decades due to the nematode's unique ability to autoinfect hosts. Immunosuppression from steroid therapy, T-lymphocytic viral (HTLV-1) infections, or a variety of underlying medical conditions may then result in dissemination and the highly lethal and infectious hyperinfection syndrome. Clinical suspicions for the condition are often not high in non-endemic areas, the diagnosis is difficult, and the incidence is increasing, particularly given recent mass population movements. Indications of infection at autopsy include gastrointestinal ulceration and haemorrhage, with pulmonary oedema, congestion, haemorrhage and diffuse alveolar damage.
Topics: Animals; Carrier State; Feces; Forensic Pathology; Hemorrhage; Humans; Immunocompromised Host; Intestinal Diseases, Parasitic; Larva; Opportunistic Infections; Pulmonary Edema; Sputum; Strongyloides stercoralis; Strongyloidiasis; Ulcer
PubMed: 30738288
DOI: 10.1016/j.jflm.2019.01.014 -
The Lancet. Respiratory Medicine Jan 2015
Review
Topics: Altitude Sickness; Brain Edema; Humans; Hypoxia; Pulmonary Edema
PubMed: 25466336
DOI: 10.1016/S2213-2600(14)70238-3 -
Revue Des Maladies Respiratoires Dec 2021Hypobaric hypoxia, the hallmark of a high altitude environment, has important physiological effects on both the cardiovascular and respiratory systems in order to... (Review)
Review
Hypobaric hypoxia, the hallmark of a high altitude environment, has important physiological effects on both the cardiovascular and respiratory systems in order to maintain a balance between oxygen demand and supply. This dynamic of acclimatization is influenced both by the level of altitude and the speed of progression, but is also very individual with a wide spectrum of responses and sensitivities. This wide range of responses is associated with nonspecific symptoms characterising acute mountain sickness and high-altitude cerebral or pulmonary oedema. This article reviews the current knowledge about both the acclimatization processes and specific diseases of high-altitude.
Topics: Acclimatization; Altitude; Altitude Sickness; Humans; Hypoxia; Pulmonary Edema
PubMed: 34782179
DOI: 10.1016/j.rmr.2020.12.007