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British Journal of Hospital Medicine... Mar 2018
Review
Topics: Acute Disease; Addison Disease; Emergencies; Humans; Hydrocortisone
PubMed: 29528741
DOI: 10.12968/hmed.2018.79.3.C34 -
Reviews in Endocrine & Metabolic... May 2003
Review
Topics: Acute Disease; Addison Disease; Adrenal Glands; Critical Care; Emergencies; Humans; Mineralocorticoids; Patient Education as Topic; Radiography
PubMed: 12766542
DOI: 10.1023/a:1022938019091 -
The Canadian Veterinary Journal = La... May 2023Primary objectives of this study were to determine presenting complaints, physical examination, clinicopathologic findings, and hospitalization time of dogs with...
OBJECTIVE
Primary objectives of this study were to determine presenting complaints, physical examination, clinicopathologic findings, and hospitalization time of dogs with spontaneous hypoadrenocorticism presenting with critical disease; and to compare those end points to dogs with a more stable presentation. Secondary objectives were to evaluate the shock index and to identify precipitating stressors.
ANIMALS
Eighty-four dogs at the Western College of Veterinary Medicine between 1998 and 2018 were included.
PROCEDURE
Data were retrieved from the medical records.
RESULTS
Collapse and depression were more common among critically ill dogs. Hyperlactatemia was rare despite a diagnosis of hypovolemic shock, and a shock index was ineffective in this patient subset. Isosthenuria, total hypocalcemia, and more severe acidosis were more common ( < 0.05) in critical dogs. Owner separation was the most common precipitating stressor.
CONCLUSION AND CLINICAL RELEVANCE
We concluded that the critical Addisonian dog has unique characteristics that may aid in early disease identification.
Topics: Dogs; Animals; Dog Diseases; Adrenal Insufficiency; Acidosis
PubMed: 37138712
DOI: No ID Found -
Der Anaesthesist Jun 2012An Addisonian crisis marks an acute adrenocortical failure which can be caused by decompensation of a chronic insufficiency due to stress, an infarct or bleeding of the...
An Addisonian crisis marks an acute adrenocortical failure which can be caused by decompensation of a chronic insufficiency due to stress, an infarct or bleeding of the adrenal cortex and also abrupt termination of a long-term glucocorticoid medication. This article reports the case of a 25-year-old patient with Crohn's disease who suffered an Addisonian crisis with hypotension, hyponatriemia and hypoglycemia during an emergency laparotomy after he had terminated prednisolone medication on his own authority. This necessitated an aggressive volume therapy in addition to an initial therapy with 100 mg hydrocortisone, 8 g glucose and a continuous administration of catecholamines. Under this treatment regimen hemodynamic stabilization was achieved. Reduction of the administration of hydrocortisone after 3 days resulted in cardiovascular insufficiency which required an escalation of the hydrocortisone substitution.
Topics: Addison Disease; Adrenal Cortex Function Tests; Adult; Anesthesia; Anti-Inflammatory Agents; Blood Volume; Catecholamines; Critical Care; Critical Illness; Crohn Disease; Fluid Therapy; Humans; Hydrocortisone; Intraoperative Complications; Laparotomy; Male; Prednisolone; Water-Electrolyte Imbalance
PubMed: 22695777
DOI: 10.1007/s00101-012-2033-1 -
Journal of Gastrointestinal Cancer Jun 2013
Review
Topics: Addison Disease; Adenocarcinoma; Adrenal Gland Neoplasms; Antimetabolites, Antineoplastic; Capecitabine; Deoxycytidine; Fluorouracil; Humans; Male; Middle Aged; Rectal Neoplasms
PubMed: 22890387
DOI: 10.1007/s12029-012-9426-x -
Journal of Clinical Medicine May 2015We report a case of a previously undiagnosed panhypopituitarism initially presenting as a full-blown Addisonian crisis with hypoglycemia, hyponatremia, hypotension and...
We report a case of a previously undiagnosed panhypopituitarism initially presenting as a full-blown Addisonian crisis with hypoglycemia, hyponatremia, hypotension and neuropsychological symptoms, more than 30 years after a severe traumatic brain injury (TBI). The patient also displayed clearly visible pathognomonic clinical signs of long-standing pituitary dysfunction. The case highlights the importance of being aware of endocrine sequelae even decades after serious TBI.
PubMed: 26239458
DOI: 10.3390/jcm4050965 -
Clinical Medicine (London, England) Mar 2020
Topics: Acute Disease; Addison Disease; Humans; Quality Improvement
PubMed: 32409395
DOI: 10.7861/clinmed.20-2-s84 -
The American Journal of Nursing Mar 1997
Topics: Acute Disease; Addison Disease; Adult; Emergencies; Emergency Nursing; Female; Humans; Nursing Assessment; Physical Exertion
PubMed: 9055760
DOI: No ID Found