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Survey of Ophthalmology 2016Acquired color vision deficiency occurs as the result of ocular, neurologic, or systemic disease. A wide array of conditions may affect color vision, ranging from... (Review)
Review
Acquired color vision deficiency occurs as the result of ocular, neurologic, or systemic disease. A wide array of conditions may affect color vision, ranging from diseases of the ocular media through to pathology of the visual cortex. Traditionally, acquired color vision deficiency is considered a separate entity from congenital color vision deficiency, although emerging clinical and molecular genetic data would suggest a degree of overlap. We review the pathophysiology of acquired color vision deficiency, the data on its prevalence, theories for the preponderance of acquired S-mechanism (or tritan) deficiency, and discuss tests of color vision. We also briefly review the types of color vision deficiencies encountered in ocular disease, with an emphasis placed on larger or more detailed clinical investigations.
Topics: Color Perception Tests; Color Vision; Color Vision Defects; Humans; Retinal Cone Photoreceptor Cells; Visual Field Tests
PubMed: 26656928
DOI: 10.1016/j.survophthal.2015.11.004 -
Albrecht Von Graefes Archiv Fur... 1972
Topics: Adolescent; Color; Color Perception Tests; Color Vision Defects; Diagnosis, Differential; Humans; Male; Methods
PubMed: 4538392
DOI: 10.1007/BF00410058 -
Journal of the Optical Society of... Feb 2012
Topics: Color Vision; Color Vision Defects
PubMed: 22330411
DOI: 10.1364/JOSAA.29.000CV1 -
The Journal of Physiology Aug 1949
Topics: Color Perception; Color Vision Defects; Humans
PubMed: 15395054
DOI: No ID Found -
Vestnik Oftalmologii 1991A total of 277 chromatopsia cases (0.09 percent of the total number) are analyzed, including erythropsia, xanthopsia, cyanopsia, chloropsia, and polychromatopsia. True...
A total of 277 chromatopsia cases (0.09 percent of the total number) are analyzed, including erythropsia, xanthopsia, cyanopsia, chloropsia, and polychromatopsia. True chromatopsia and chromatopsia due to staining of optic media of the eyes were distinguished and an attempt to explain this entoptic phenomenon made. True chromatopsias are related to depression of stimulation of the optic nerve route.
Topics: Adult; Color Vision Defects; Female; Humans; Male; Pregnancy; Pregnancy Complications
PubMed: 1858231
DOI: No ID Found -
Documenta Ophthalmologica. Advances in... Aug 1989More than half of the cases with complaints of chromatopsia had recent-onset retinal pathology. Erythropsia due to bright (sun-)light is a relatively common finding in...
More than half of the cases with complaints of chromatopsia had recent-onset retinal pathology. Erythropsia due to bright (sun-)light is a relatively common finding in aphakia and pseudophakia. UV-coated intraocular lenses do not provide complete protection. Cerebrovascular chromatopsia usually occurs in transient attacks.
Topics: Aged; Aphakia; Color Vision Defects; Humans; Lenses, Intraocular; Light; Male; Retina; Retinal Detachment; Retrospective Studies
PubMed: 2625099
DOI: 10.1007/BF00153507 -
Nature Methods Jun 2011
Topics: Color; Color Vision; Color Vision Defects; Humans; Reading; Sensory Aids; Software
PubMed: 21774112
DOI: 10.1038/nmeth.1618 -
The British Journal of Ophthalmology Jan 2016The cone dysfunction syndromes are a heterogeneous group of inherited, predominantly stationary retinal disorders characterised by reduced central vision and varying... (Review)
Review
The cone dysfunction syndromes are a heterogeneous group of inherited, predominantly stationary retinal disorders characterised by reduced central vision and varying degrees of colour vision abnormalities, nystagmus and photophobia. This review details the following conditions: complete and incomplete achromatopsia, blue-cone monochromatism, oligocone trichromacy, bradyopsia and Bornholm eye disease. We describe the clinical, psychophysical, electrophysiological and imaging findings that are characteristic to each condition in order to aid their accurate diagnosis, as well as highlight some classically held notions about these diseases that have come to be challenged over the recent years. The latest data regarding the genetic aetiology and pathological changes observed in the cone dysfunction syndromes are discussed, and, where relevant, translational avenues of research, including completed and anticipated interventional clinical trials, for some of the diseases described herein will be presented. Finally, we briefly review the current management of these disorders.
Topics: Color Vision Defects; Genotype; Humans; Phenotype; Retinal Cone Photoreceptor Cells; Retinal Diseases; Syndrome
PubMed: 25770143
DOI: 10.1136/bjophthalmol-2014-306505 -
The Lancet. Neurology Jun 2002
Topics: Animals; Color Perception; Color Vision Defects; Humans; Retinal Pigments
PubMed: 12849524
DOI: 10.1016/s1474-4422(02)00051-0 -
Eye (London, England) May 2010Colour vision deficiency is one of the commonest disorders of vision and can be divided into congenital and acquired forms. Congenital colour vision deficiency affects... (Review)
Review
Colour vision deficiency is one of the commonest disorders of vision and can be divided into congenital and acquired forms. Congenital colour vision deficiency affects as many as 8% of males and 0.5% of females--the difference in prevalence reflects the fact that the commonest forms of congenital colour vision deficiency are inherited in an X-linked recessive manner. Until relatively recently, our understanding of the pathophysiological basis of colour vision deficiency largely rested on behavioural data; however, modern molecular genetic techniques have helped to elucidate its mechanisms. The current management of congenital colour vision deficiency lies chiefly in appropriate counselling (including career counselling). Although visual aids may be of benefit to those with colour vision deficiency when performing certain tasks, the evidence suggests that they do not enable wearers to obtain normal colour discrimination. In the future, gene therapy remains a possibility, with animal models demonstrating amelioration following treatment.
Topics: Color Vision Defects; Eyeglasses; Female; Filtration; Genetic Therapy; Humans; Male
PubMed: 19927164
DOI: 10.1038/eye.2009.251