-
Tierarztliche Praxis. Ausgabe G,... Apr 2022In a herd of 40 cattle of the Holstein-Friesian breed, 6 animals were euthanized for severe pruritus, fever and skin lesions. Pathomorphological examination of 3 animals...
In a herd of 40 cattle of the Holstein-Friesian breed, 6 animals were euthanized for severe pruritus, fever and skin lesions. Pathomorphological examination of 3 animals revealed multisystemic granulomatous inflammation involving multinuclear giant cells. The content of vetch in the feed used and the characteristic histology led to the diagnosis of vetch poisoning.
Topics: Animals; Cattle; Cattle Diseases; Plant Breeding; Plant Poisoning; Vicia
PubMed: 35523189
DOI: 10.1055/a-1779-3265 -
Australian Veterinary Journal Aug 2006
Review
Topics: Animals; Australia; Cattle; Cattle Diseases; Glycolipids; Horse Diseases; Horses; Nematoda; Plant Poisoning; Sheep; Sheep Diseases
PubMed: 16911226
DOI: 10.1111/j.1751-0813.2006.00019.x -
Journal of Medical Toxicology :... Apr 2024
Topics: Humans; Nerium; Plant Poisoning; Foodborne Diseases
PubMed: 38324189
DOI: 10.1007/s13181-024-00993-3 -
Annals of Emergency Medicine Oct 1990We report the cases of a child who ingested two or more castor beans and two adults who each ingested four beans. All three patients developed severe gastroenteritis and... (Review)
Review
We report the cases of a child who ingested two or more castor beans and two adults who each ingested four beans. All three patients developed severe gastroenteritis and recovered without sequelae after receiving IV fluids. The literature contains reports of 424 cases of castor bean intoxication. Symptoms of intoxication include acute gastroenteritis, fluid and electrolyte depletion, gastrointestinal bleeding, hemolysis, and hypoglycemia. Delayed cytotoxicity has not been reported. Of the 424 patients, 14 died (mortality rates: 8.1% of untreated and 0.4% of treated). Deaths were due to hypovolemic shock. Recommended treatment for asymptomatic patients who have chewed one or more raw beans is emergency department evaluation, gastric decontamination, administration of activated charcoal, observation until four to six hours after ingestion, and discharge instructions to return if symptoms develop. After decontamination and activated charcoal, symptomatic patients require hospitalization for treatment with IV fluids, supportive care, and monitoring for hypoglycemia, hemolysis, and complications of hypovolemia. Monitoring for delayed cytotoxicity is unnecessary. Castor beans and their dust are highly allergenic and may cause anaphylaxis.
Topics: Adult; Ricinus communis; Charcoal; Child, Preschool; Fluid Therapy; Gastroenteritis; Humans; Male; Plant Poisoning; Plants, Toxic
PubMed: 2221525
DOI: 10.1016/s0196-0644(05)81525-2 -
The Veterinary Record Jul 1983
Topics: Animals; Female; Plant Poisoning; Sheep; Sheep Diseases
PubMed: 6880001
DOI: 10.1136/vr.113.1.23-a -
Australian Veterinary Journal Jul 2007
Review
Topics: Animal Feed; Animals; Cattle; Cattle Diseases; Pennisetum; Plant Poisoning; Risk Factors; Seasons
PubMed: 17615037
DOI: 10.1111/j.1751-0813.2007.00168.x -
Tierarztliche Praxis. Ausgabe G,... 2011Pyrrolizidine alkaloids belong to a class of phytotoxins which are present in more than 6000 plant species. The disease seneciosis in farm animals represents the severe... (Review)
Review
Pyrrolizidine alkaloids belong to a class of phytotoxins which are present in more than 6000 plant species. The disease seneciosis in farm animals represents the severe poisoning by pyrrolizidine alkaloids from plants of the genus Senecio. This form of poisoning has been known since the end of the 19th century in Germany, the USA, Canada and New Zealand, and is mainly caused by Senecio jacobaea and related Senecio spp. in farm animals, including poultry. Animal poisoning by pyrrolizidine alkaloids is of worldwide importance. In Germany poisoning of horses and cattle by Senecio jacobaea, which was earlier named Schweinsberg disease, is of renewed relevance for veterinary medicine. The disease occurs almost entirely as a consequence of chronic poisoning and in general ends fatally. The ultimate cause is the formation of toxic metabolites of pyrrolizidine alkaloids in the liver, and their covalent binding to nucleic acids and proteins leading to liver cirrhosis. Because many pyrrolizidine alkaloids possess mutagenic, and a few also carcinogenic properties, European and international authorities are concerned about possible residue levels in food of animal origin. The review addresses in its first part several aspects, being the occurrence, the chemistry, and the toxicology of pyrrolizidine alkaloids as well as animal intoxications by poisonous plants. In the second part (46) clinical characteristics of animal seneciosis, the therapeutic interventions, the significant species differences and a critical assessment of so-called nontoxic amounts of Senecio plants in animal fodder with reference to cumulative lethal toxin doses are presented.
Topics: Animals; Animals, Domestic; Plant Poisoning; Pyrrolizidine Alkaloids; Senecio
PubMed: 22138830
DOI: No ID Found -
Journal of Natural Toxins Oct 1999
Review
Topics: Animals; Animals, Domestic; Cattle; Oxalates; Plant Poisoning; Plants, Toxic; Sheep; United States
PubMed: 10591042
DOI: No ID Found -
Australian Veterinary Journal Sep 2008
Topics: Animals; Dog Diseases; Dogs; Phylogeny; Plant Poisoning; Plants, Toxic
PubMed: 18782414
DOI: 10.1111/j.1751-0813.2008.00344.x -
The Veterinary Record Jul 1999
Topics: Animals; Horse Diseases; Horses; Incidence; Plant Poisoning; Plants, Toxic; Senecio; United Kingdom
PubMed: 10466840
DOI: No ID Found